Providing Atorfin works on several levels to control myopia. First, atropine, which blocks a role Accommodative
\nThe progress of myopia. As an antagonist Moskrini, it slows down the length of the eye [15; 18], also affects
\nThe release of dopamine cells, and therefore affects the signals that control the rate of growth Hrtinliim eye [19]. Also, atropine absorbed
\nSystemic, can also inhibit the secretion of growth hormone, and inhibit the growth of the eye [20].
\nA study that examined 212 children in care and glasses Bifokaliot atropine for an average of 3.5 years showed that the rate of growth of short
\nVision was smaller in patients compared to a control group of 21, including the treatment of combined wear glasses Atorfin
\nMoltifokliim introduced greater efficiency [22]. Multicenter studies (MIT and ATOM) have also shown the efficacy of atropine
\nConcentration of 1%, after years of progress fell significantly throughout the eye compared to the control group.<\/p>\n
Other studies have examined different concentrations of atropine from 0.05% to 1%. They all reported that treatment of atropine, partly combined
\nB-Fokliim glasses, slowed the progression of myopia. However participants reported numerous complaints of blurred vision
\nAnd blinding [18; 23; 24].
\nDespite the success of the treatment have not been determined atropine treatment concentrations and duration of treatment. In addition to atropine There are two broad effects
\nThe first is prolonged paralysis Accommodative and the second is to expand the pupil and these require optical correction and masking nearly blind
\nThe study’s match. The first is paralysis Accommodative, and the second is pupil dilation. Accommodative paralysis requires optical correction
\nClose study participants. Expanded pupil and optical correction relative influence on the ability to screen the blind study and hence the quality
\nThe experiments may be affected [18]. In addition, the effect of atropine long-term eye health including early presbyopia, damage
\nRetina and cataract, not yet clear [14; 16; 19; 20; 23; 24].<\/p>\n
Encouraging results come just use low concentrations of atropine. Atropine treatment at a concentration of 0.01% does not factor in the effects
\nVision and clinical efficacy results showed high tolerance with respect to higher concentrations [25]. Atropine at a concentration of 0.01%
\nThere is a negligible effect on Accommodative and pupil size and therefore does not affect the quality and visual acuity closely [25].<\/p>\n","protected":false},"excerpt":{"rendered":"
0722-6785858TEL\u00a0 \u00a0Contact The Center of Keratconus-Dr Nir Erdinest Providing Atorfin works on several levels to control myopia. First, atropine, which blocks a role Accommodative The progress of myopia. As an antagonist Moskrini, it slows down the length of the eye [15; 18], also affects The release of dopamine cells, and therefore affects the signals that […]<\/p>\n","protected":false},"author":1,"featured_media":0,"parent":0,"menu_order":0,"comment_status":"open","ping_status":"open","template":"","meta":{"_joinchat":[],"footnotes":""},"class_list":["post-3479","page","type-page","status-publish","hentry"],"yoast_head":"\n